Melatonin in Mitochondria: Mitigating Clear and Present Dangers (2020): Difference between revisions

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Reiter RJ, Ma Q, Sharma R. Melatonin in Mitochondria: Mitigating Clear and Present Dangers. Physiology (Bethesda). 2020 Mar 1;35(2):86-95. doi: 10.1152/physiol.00034.2019. PMID: 32024428.
Reiter RJ, Ma Q, Sharma R. Melatonin in Mitochondria: Mitigating Clear and Present Dangers. Physiology (Bethesda). 2020 Mar 1;35(2):86-95. doi: 10.1152/physiol.00034.2019. PMID: 32024428.
[[Category:Research Papers]]
[[Category:Research Papers]]
[[Category:Reviews]]
[[Category:Mitochondria]]
[[Category:Mitochondria]]

Revision as of 15:15, 23 March 2023

Explanation

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Highlights

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Related Studies

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Official Abstract

In cancer cells, glucose is primarily metabolized to pyruvate and then to lactate in the cytosol. By allowing the conversion of pyruvate to acetyl-CoA in mitochondria, melatonin reprograms glucose metabolism in cancer cells to a normal cell phenotype. Acetyl-CoA in the mitochondria also serves as a necessary co-factor for the rate-limiting enzyme in melatonin synthesis, thus ensuring melatonin production in mitochondria of normal cells.

Publication Details

Authors

Russel J. Reiter, Qiang Ma, and Ramaswamy Sharma

Journal

Physiology

PubMed

https://pubmed.ncbi.nlm.nih.gov/32024428/

Full Text

https://journals.physiology.org/doi/full/10.1152/physiol.00034.2019

Citation

Reiter RJ, Ma Q, Sharma R. Melatonin in Mitochondria: Mitigating Clear and Present Dangers. Physiology (Bethesda). 2020 Mar 1;35(2):86-95. doi: 10.1152/physiol.00034.2019. PMID: 32024428.